The symptoms of nephritic syndrome varying depending on the type of amboss. A characteristic symptom is edema, a fluid accumulation in the body that can produce a puffy appearance. Patients may have oliguria, which is low urine output (less than 500 ml in 24 hours). Hematuria, on the other hand, is the presence of blood in the urine. This blood is either micro or macrohematuria and is classified as visible or unseen blood.
Membrane nephropathy is the most common cause of nephritic syndrome in adults. It often develops following streptococcal skin or throat infections. Group A beta hemolytic streptococci are nephrogenic strains. Ninety percent of patients show a previous infection with these types of bacteria. The M protein on the bacterial cell wall is what determines the type of nephrogenic strains.
The underlying cause of nephritic syndrome can be identified through a renal biopsy. While guidelines for renal biopsy are controversial, most treatments focus on prevention and treatment of complications such as infection, hyperlipidemia, and venous thrombosis. For those with edema, diuretics can reduce fluid and swelling. The diagnosis of nephritic syndrome is critical to ensuring a full recovery.
Nephritic syndrome is a pathological condition of the kidneys. A breakdown in the glomerular filtration barrier results in large amounts of albumin and red blood cells in urine. In severe cases, patients may exhibit dysmorphic RBCs, known as acanthocytes. When RBCs deform, they can form casts. These casts are encased in Tamm-Horsfall protein, which is secreted by the renal tubular cells and excreted in the urine.